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Macrophage studies demonstrate that GHK-Cu administration shifts these cells from a pro-inflammatory M1 phenotype to a tissue-repairing M2 phenotype, increasing their production of growth factors including vascular endothelial growth factor (VEGF) and platelet-derived growth factor (PDGF) by 40-60%

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221,591 Crystal structures of both apo- and molybdate-bound forms of ModE 597 reveal that the molybdate binding to the C-terminal domain alters the relative orientation of the N-terminal DNA binding domains from each protomer, allosterically activating the ModE homodimer to bind DNA (Figure 21C)